Factors associated with myocardial damage during transcutaneous coronary intervention in patients with acute coronary syndrome
- Authors: Maksimov N.I.1,2, Semenyuta V.V.2
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Affiliations:
- Republican Clinical Diagnostic Center
- Izhevsk State Medical Academy
- Issue: Vol 37, No 1 (2020)
- Pages: 27-34
- Section: Original studies
- URL: https://ogarev-online.ru/PMJ/article/view/18787
- DOI: https://doi.org/10.17816/pmj37127-34
- ID: 18787
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Abstract
Aim. To establish the factors, associated with myocardial damage while performing transcutaneous coronary intervention.
Materials and methods. The retrospective analysis included 255 patients, distributed by the dynamics of creatine phosphokinase-MB in the blood after the surgery.
Results. Myocardial damage while performing transcutaneous coronary intervention is interconnected with the period from the onset of symptoms to the surgery, initial levels of cardiac-specific markers, localization and degree of manifestation of coronary artery stenoses and endovascular interventions implemented.
Conclusions. As a result, the factors associated with myocardial damage while performing transcutaneous coronary intervention were revealed and their concrete boarders were determined.
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##article.viewOnOriginalSite##About the authors
N. I. Maksimov
Republican Clinical Diagnostic Center; Izhevsk State Medical Academy
Email: hosp-therap@igma.udm.ru
Head of the Department of Hospital Therapy, Doctor of Medical Science, Professor
Russian Federation, Izhevsk
V. V. Semenyuta
Izhevsk State Medical Academy
Author for correspondence.
Email: semenyuta0@gmail.com
6th year student at the medical faculty
Russian Federation, IzhevskReferences
- Акинина С.А. Сравнительное прогностическое значение креатинфосфокиназы МВ-фракции и тропонина Т при диагностике перипроцедурного повреждения миокарда. Российский кардиологический журнал 2012; 3: 19–24.
- Копица Н.П., Вишневская И.Р. Феномен «No-reflow» – современный взгляд на проблему. Место блокаторов гликопротеиновых IIb/IIIa рецепторов в предупреждении данного состояния. Вестник ХНУ им. В.Н. Каразина. Серия: Медицина 2011; 22: 975.
- Крылов А.Л., Сыркина А.Г., Алексеева Я.В., Демьянов С.В., Баев А.Е., Марков В.А., Рябов В.В. Двухэтапная реваскуляризация у пациентов с острым инфарктом миокарда и массивным тромбозом коронарной артерии. Кардиология 2019; 2: 5–9.
- Петрищев Н. Н., Шляхто Е. В., Галагудза М.М. Новые способы защиты миокарда от ишемического и реперфузионного повреждения: молекулярные механизмы и перспективы клинического применения. Кардиология 2007; 5: 179–184.
- Стафеев Ю.С., Меньшиков М.Ю., Ткачук В.А., Парфенова Е.В. Роль макрофагов в репарации миокарда после повреждения и перспективы метаболического перепрограммирования иммунных клеток в целях регуляции постинфарктного восстановления миокарда. Кардиология 2017; 12: 53–59.
- Abbo K.M., Dooris M., Glazier S., O'Neill W.W., Byrd D., Grines C.L., Safian R.D. Features and outcome of no-reflow after percutaneous coronary intervention. The American journal of cardiology 1995; 75: 778–782.
- Abdelmequid A.E., Topol E.J. The myth of myocardial ‘infarctlet’ during percutaneous coronary revascularization procedures. Circulation 1996; 94: 3369–3375.
- Cavallini C., Savonitto S., Violini R., Arraiz G., Plebani M., Olivari Z., Rubartelli P., Battaglia S., Niccoli L., Steffenino G., Ardissino D. Impact of the elevation of biochemical markers of myocardial damage on long term mortality after percutaneous coronary intervention: results of the CK MB and PCI study. European heart journal 2005; 26: 1494–1498.
- Cuculi F., Lim C.C., Banning A.P. Periprocedural myocardial injury during elective percutaneous coronary intervention: is it important and how can it be prevented? Heart 2010; 96: 736–740.
- Cutlip D.E., Windecker S., Mehran R., Boam A., Cohen D.J., van Es G.A., Steg P.G., Morel M.A., Mauri L., Vranckx P., McFadden E., Lansky A., Hamon M., Krucoff M.W., Serruys P.W. Clinical end points in coronary stent trials: a case for standardized definitions. Circulation 2007; 115: 2344–2351.
- Jaffe R., Charron T., Puley G., Dick A., Strauss B.H. Microvascular obstruction and the no-reflow phenomenon after percutaneous coronary intervention. Circulation 2008; 117: 3152–3156.
- Niccoli G., Burzotta F., Galiuto L., Crea F. Myocardial No-Reflow in Humans. Journal of the American College of Cardiology 2009; 54: 281–292.
- Ottervanger J.P., Armstrong P., Barnathan E.S., Boersma E., Cooper J.S., Ohman E.M., James S., Topol E., Wallentin L., Simoons M.L. Long-Term Results After the Glycoprotein IIb/IIIa Inhibitor Abciximab in Unstable Angina. Circulation 2003; 103: 437–442.
- Pasceri V., Patti G., Nusca A., Pristipino C., Richichi G., Di Sciascio G. Randomized Trial of Atorvastatin for Reduction of Myocardial Damage During Coronary Intervention: Results From the ARMYDA Study. Circulation 2004; 110: 674–678.
- Pinto D.S., Gibson C.M., Wykrzykowska J.J. Ischemic reperfusion injury of the heart. UpToDate Inc 2009, available at: http://www. http://uptodate.com/contents/ischemic-reperfusion-injury-oftheheart.
- Vranckx P., Cutlip D.E., Mehran R., Kint P.P., Silber S., Windecker S., Serruys P.W. Myocardial infarction adjudication in contemporary all comer stent trials: balancing sensitivity and specificity. EuroIntervention 2010; 5 (7): 871–874.
- Secemsky E.A., Ferro E.G., Rao S.V., Kirtane A., Tamez H., Zakroysky P., Wojdyla D., Bradley S.M., Cohen D.J., Yeh R.W. Association of Physician Variation in Use of Manual Aspiration Thrombectomy With Outcomes Following Primary Percutaneous Coronary Intervention for ST-Elevation Myocardial Infarction: The National Cardiovascular Data Registry CathPCI Registry. JAMA Cardiology 2019; 4 (2): 110–118.
- Thygesen K., Alpert J.S., Jaffe A.S., Chaitman B.R., Bax J.J., Morrow D.A., White H.D. Fourth universal definition of myocardial infarction. European heart journal 2019; 40 (3): 237–269.
- Wu A.H., Apple F.S., Gibler W.B., Jesse R.L., Warshaw M.M., Valdes R.Jr. National Academy of Clinical Biochemistry Standards of Laboratory Practice: Recommendations for the use of cardiac markers in coronary artery diseases. Clinicalchemistry 1995; 45: 1104–1121.
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