The structural-functional characteristics of myocardium in the experimental crush syndrome

Abstract

The investigation of the pathogenesis mechanisms and the morphological manifestations of crush syndrome are still actual. There is still a persistent likelihood of occurrence of the situations that can possibly lead to the given pathology: the lesions as a rule gain an incremental character, and what is more, the developing crush syndrome in such cases occurs in its heaviest form. The exploited model of the experiment has enabled to mitigate CS; the lighter form of the syndrome has made the development of a more sustainable model of CS possible, which is proved by the morphological changes of myocardium.